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Revise Entire Ortho in Just 70 mins | NEET PG | INICET | FMGE | Dr. Apurv Mehra

1h 9m 36s10,747 words1,690 segmentsEnglish
“This is the only condition in orthopedics where you can fuse a joint because life is movement.”
▶ 42:43 · HEAR IT IN THE VIDEO

FULL TRANSCRIPT

0:00

Let's have a look at entire orthopedics

0:06

in

0:08

100 lines. Let's go.

0:12

Orthopedics means straight child.

0:17

Nicholas gave this word called as the

0:20

father of orthopedics.

0:22

The second father is Thomas who gave

0:26

Thomas splint and Thomas test for hip

0:31

flexion deformities.

0:33

Chanlays is called as the father of

0:36

arroplasty.

0:38

When you look at X-rays, it can show you

0:40

the bone. It can show you the soft

0:42

tissue planes. It can show you the

0:44

cortex in the bone. It can show you the

0:46

marrow. What it cannot show you is the

0:49

cartilage. When you look at the CT scan,

0:52

it can show you the calcifications. It

0:54

can show you the cortex. CT scan C,

0:56

cortex C, calcification C. But it's

1:00

rarely an investigation. In orthopedics,

1:02

if you want to choose an investigation,

1:05

go for MRI because it can show you the

1:08

marrow, it can show you the soft tissue,

1:10

it can show you the cartilage. Cartilage

1:12

is not seen on X-rays, is not picked up

1:14

on CT scan, it's picked up on MRI. When

1:17

I look at tumors and infection which are

1:20

usually the scenarios given to you for

1:22

diagnosis, biopsy or tissue is the gold

1:26

standard. Radiology is suggestive.

1:29

Tissue is diagnostic. When I look at

1:32

metastasis, when I look at metastasis,

1:34

PET scan is the investigation, the

1:37

radological investigation of choice. But

1:39

bone scan is preferred in osteoplastic

1:41

meds. And can you guess what causes

1:44

osteoplastic meds? Prostate. The

1:46

prostate is known for causing

1:48

osteoplastic metastasis. Perio reaction

1:50

came in neat PG 2024 paper 1 paper two

1:54

when they show you any perioile reaction

1:56

which is single layer solid perryio

1:58

reaction is benign bone tumor or acute

2:00

osteomalitis when wide zone of activity

2:02

31 they ask you number one sunray

2:06

appearance usually osteocaroma

2:09

codman's triangle usually osteocaroma

2:13

onion peel appearance usually evening

2:15

saroma but the location of the tumor is

2:17

more important than these radological

2:20

images. So think about e-wings in the

2:23

middle of a bone diaphasis and

2:25

osteocaromas

2:27

on the lower end or the upper end the

2:29

metaphysis. Remember in osteomalitis

2:32

you will get an X-ray done first

2:35

followed by MRI but MRI shows you

2:37

changes within 6 to 12 hours whereas

2:40

X-rays will show you loss of soft tissue

2:43

plane after 24 hours and perostal

2:46

reactions on day 7 to day 10. When we

2:49

look at the infections you know the king

2:52

of orthopedic is stefallocazorius

2:55

osteomalitis comes through blood

2:57

hematogenous is in the metaphysis and

3:01

when they ask you the organism remember

3:04

that staporius is the king exception

3:06

that you should remember is cle cell

3:08

anemia salmonella and foot is something

3:12

where you should think about an ivas you

3:14

should think about sudinas

3:17

osteomalitis when It's chronic which is

3:19

one of the most common invest the images

3:21

shown to you in orthopedics. Think about

3:24

sequestrum the dead bone which is

3:25

diagnostic and when you remove the

3:27

sequestrum the live bleeding bone comes

3:30

that's called as a paprika sign.

3:32

Remember multif focal oatis that is

3:34

osteolitis involving the multiple bones

3:37

right safo syndrome is number one safo

3:40

syndrome is a misnomer it is actual

3:42

osteitis but if they ask you which is

3:45

multif focal oitis choose safo syndrome.

3:48

Second common is cle cell anemia with

3:49

salmonella infections. Faber flexion

3:53

abideduction externation

3:56

of the lower limb is a position of

3:59

increased capacity of the hip joint

4:01

which is seen in sinovitis

4:04

infection anterior dislocation and when

4:07

there is ilotibial band contraurs. Faber

4:10

is usually associated with lengthening.

4:13

Opposite of it flexion, adduction,

4:16

interotation that is far is indicative

4:19

of arthritis or posterior dislocation.

4:22

So posterior dislocation more common

4:24

than arthritis. Trauma posterior

4:26

dislocations are much more common than

4:28

anterior dislocation. That is why faber

4:30

anterior dislocation is a cause later

4:32

but in firer posterior dislocation is

4:34

number one cause. And remember fer

4:37

shortening is there. Faber lengthening

4:40

is there. If you look at the infections

4:43

of the joints, they are surgical

4:44

emergencies called as septic arthritis.

4:47

Organism is staporious. Less than 5

4:49

years, toxic child. Compared to it,

4:52

transient sinoitis, temporary

4:54

proliferation of sinovium, age group is

4:57

6 to 12 years, nonoperative management.

5:00

Infections of the hand. The most common

5:03

infection of the hand is the infection

5:05

of the nail bed called as peronicia

5:07

organism staphorius. Infections in the

5:11

felon or vitlo is the infection of pulse

5:14

space of finger. The organism again the

5:16

king of orthopedic staff orius. When you

5:19

look at a chronic disease we live in

5:21

India. We talk about tuberculosis.

5:23

Tuberculosis of bone and joint is

5:25

usually secondary. Comes from the lungs

5:27

or the lymph nodes. Tuberculosis of bone

5:30

and joint is just like Shah Ruk Khan SRK

5:33

SHK spine then hip the knee. TB spine

5:37

also called as spot spine or cockpine

5:39

will never involve a single vertebrae.

5:41

It will involve two contiguous vertebrae

5:44

with the intervening disc called as

5:46

paradiscal involvement. The disease is

5:49

anterior to the neural tissue that is

5:51

the spinal cord as you can see in this

5:52

image and the disease rarely involve the

5:55

posterior elements that is the

5:57

involvement of the facet joints or the

6:00

spinus process is rarest in TB spine

6:03

remember tuberculosis spine is treated

6:05

by middle path regimen what means is

6:07

anti-tubacular therapy where ifin is the

6:10

most important drug plus rest to that

6:13

body part and surgery when indicated

6:15

what does that mean it means that you

6:17

will operate only when required But not

6:18

that you will never operate. Not that

6:20

you will always operate. Remember in TB

6:23

spine right because of the parispinal

6:26

muscle spasm the loss of curvature of

6:28

the spine is seen much earlier than the

6:31

reduced space. But the tuberculosis of

6:33

other parts of the body where the blood

6:35

supply becomes more because infections

6:37

get inflammation. Inflammation gets more

6:40

blood supply. There is osteopenia around

6:42

the joint called as per articular

6:44

osteopenia. Remember this when we look

6:47

at tuberculosis the knee joint right

6:50

there can be a triple deformity which is

6:52

posterior subluxation externation and

6:54

flexion whereas TB hip has got five

6:57

stages stage one remember faber fer

7:01

faber sinoitis lengthening good

7:04

prognosis stage two and three are stages

7:07

of arthritis there is flexion adduction

7:10

introduction in stage two less than 1 cm

7:13

shortening which is not clinically

7:14

appreciable

7:15

Stage three, late or advanced arthritis.

7:18

Whenever they mention shortening, it's

7:20

automatic. They're talking about more

7:21

than an inch. Forget about more than a

7:23

centimeter. So mark stage three. Stage

7:26

four, the acetablum is destroyed. The

7:28

femoral head is destroyed. The destroyed

7:31

femoral head migrates from one corner

7:32

estab to other called as wandering

7:35

establum. It should be better called as

7:37

wandering femoral head. But it's a

7:38

misnomer. And finally stage five is

7:41

fusion of the joint by fibrous tissue

7:44

called as fibrous ankylosis. Friends

7:47

arthrodesis

7:49

is surgical fusion of a joint the

7:51

surgeon fuses.

7:53

Ankyosis

7:54

is pathological fusion of a joint. The

7:57

disease fuses. The disease will not know

8:00

how much to fuse. If it fuses completely

8:02

is bony ankylosis painless. If it fuses

8:05

incomplete fibrous fibbrus as F pain

8:08

painful ankylosis TB spine bony

8:11

ankylosis everywhere else tuberculosis

8:15

will cause fibrokenyosis.

8:17

Bony fusion that is bony ankylosis is

8:20

solid.

8:22

Solid is painless.

8:24

S for solid. S for septic arthritis. So

8:28

bony ankylosis most common cause is

8:31

septic arthritis. S for spine. S for

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